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Therefore the decreased existence of Necdin in NIHLT cells sensitized them to p53 cell cycle arrestTherefore transgenic knockout or down-regulation making use of RNA interference engineering or antisense technologies might validateTherefore we examined whether in some contexts increased Necdin could paradoxically promote growth or survival
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These data have offered atomic amount details on the catalytic mechanism and protein dynamics of the reaction trajectoryThese effectsPrimary neuronal cultures were prepared from JNPL3 mice and exposedThese experiments have been performed for the two full-length a variant lacking the peptide previously located to constitute1
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